Documented experiences of the second wind go back no less than a hundred years, when it was taken to be a generally held reality of exercise. Regardless of whether or not the patient experiences symptoms of muscle ache, muscle fatigue, or cramping, the phenomenon of second wind having been achieved is demonstrable by the sign of an increased heart price dropping whereas maintaining the identical velocity on the treadmill. Inactive patients experienced second wind, demonstrated by means of relief of typical signs and the sign of an increased coronary heart price dropping, injury prevention with movesmethod while performing low-moderate aerobic exercise (strolling or brisk walking). For the frequently active patients, it took more strenuous exercise (very brisk walking/jogging or bicycling) for them to experience both the standard signs and relief thereof, along with the signal of an increased coronary heart rate dropping, restore mobility program demonstrating second wind. As such, restore mobility program those with muscle glycogenoses don't must do extended exercise to experience "hitting the wall". In experienced athletes, "hitting the wall" is conventionally believed to be as a result of body's glycogen shops being depleted, with "second wind" occurring when fatty acids develop into the predominant supply of power.

The delay between "hitting the wall" and "second wind" occurring, has to do with the gradual velocity at which fatty acids sufficiently produce ATP (power)